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ASD varies in degrees of severity, occurring in 1% of the population, and its prevalence has been increasing during the last three decades ( FMR1 gene sequence on the chromosome X that results in fragile X syndrome (FXS) or fragile X-premutation tremor/ataxia syndrome (FXTAS) are among the leading genetic causes of ASD ( It has been recently proposed that a significant number of ASD cases could be associated with various metabolic abnormalities, some of them identifiable only through untargeted metabolomic profiling, simultaneously opening additional space for therapeutic attempts ( Inborn Errors of Metabolism Clinical symptoms deriving from central nervous system (CNS) occur in more than 50% of patients with inborn errors of metabolism (IEM)

10.1007/s40200-022-01053-9 118 ZhangB.ChenX.RuF.GanY.LiB.XiaW.et al (2021)

Some studies in rodents have added KYNA at massive doses (well over 100 mg/kg, getting into the millimolar range in serum/plasma), seemingly without ill effects (indeed sometimes with protective effects)

Table 1 Multi-targeted mechanisms of hepatoprotection