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It acts as a cofactor for two important enzymes: methionine synthase, which is involved in DNA production and the metabolism of homocysteine (an amino acid that may be linked to cardiovascular risk when elevated), and methylmalonyl-CoA mutase, which is essential for energy production from fats and proteins

However, because IKE decreased cystine uptake and GSH/GSSG levels and increased ROS, yet did not induce a marked increase in cell death when used alone, our data suggests that a dominant ferroptotic cell death induction is unlikely in our experimental settings

Am J Trop Med Hyg 86:464469

It's basically a fatty acid delivery system (~75-85% oleic acid) plus some minor extras like squalene, tocopherols, and phytosterols